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Serum homocysteine levels were measured using the enzyme cycling assay (Roche Diagnostics, Cobas e 801, Switzerland), and serum folate and vitamin B12 levels were analyzed using automated electrochemiluminescence immunoassays (Roche Diagnostics, Cobas 8000, Switzerland)

Taken together, these results suggest that SRY may negatively regulate the expression of GSTM2/3 through the accumulation of PARP1 and the promotion of its nuclear enrichment

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As a result, the choice of optimal treatment and management of clinical trials would be enhanced, enabling the analysis of known and novel therapies [1, 2]