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Conversely, substantial airway epithelial-mesenchymal transition (EMT) is typically achieved through angiogenesis (type-3 EMT), potentially elucidating the elevated lung cancer risk in individuals with COPD [75, 79]

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Depletion of glutathione and inadequate glutathione system Regulatory T-cell failure Breakdown of barrier systems NF-kB dysregulation/chronic inflammation Impaired hepatic biotransformation All of these systems are compromised to some degree in loss of chemical tolerance and management of this condition must include assessment of all these areas and address all physiological imbalances that are identified or suspected