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explores the interactions between beta-carotene/retinol and thyroid hormones, as well as the results of clinical trials investigating the relationship between beta-carotene intake and thyroid hormone levels

Experimental modulation of endothelial survival, angiogenic signaling and metabolic state reveals that intact tyrosine kinase receptor (Tie2) signaling, glycolytic competence of peritubular capillary endothelial cells, and sustained VEGF-A-vascular endothelial growth factor receptor 2 (VEGFR2) communication from injured TECs are all required to maintain endothelial identity, prevent EndMT and limit microvascular rarefaction [121]

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