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Impaired mitochondrial redox homeostasis, such as oxidative stress due to excessive ROS production and impairment of antioxidant function, can cause mitochondrial dysfunction and the onset of the cell death cascade, leading to neuronal damage (115, 116)

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Documented synergy between GHRH and GHRP pathways across multiple preclinical models consistently shows combined administration produces GH release exceeding the sum of individual effects, typically 2- to 5-fold higher than predicted from additive responses