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Translational Andrology and Urology, 5 (1), 88101

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Interestingly, in this study, each compound did not induce oxidative stress and mitochondrial damage at the maximum concentration that did not cause cell death of HUVECs, while the combination of these compounds at the same concentrations induced oxidative stress and mitochondrial outer membrane permeabilization that are below the threshold necessary to trigger apoptosis

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