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In addition to the acute injury, inflammation after stroke contributes to secondary cell injury, mediated, at least in part to NLRP3 inflammasome formation in microglia, Ms located within the perivascular space, and endothelial cells themselves (Chen et al., 2019

However, the controls exhibited sustained spasticity until the end of the experiment (day 360) while the BPC 157 rats exhibited resolved spasticity by day 15 (Fig

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