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Furthermore, treatment with Lumacaftor, a CFTR corrector facilitating the trafficking of misfolded F508del-CFTR protein, led to a reduction of superoxide levels in CFBE41o-cells, suggesting that while Lumacaftor primarily addresses CFTR protein processing, its therapeutic efficacy might also stem from mitigating oxidative damage and subsequent inflammatory responses (de Bari et al., 2018)

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doi: 10.1037/a0023034 71 GurneyS